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ePublished: 21 Oct 2016
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J Renal Inj Prev. 2017;6(1): 12-15.
doi: 10.15171/jrip.2017.02
PMID: 28487865
PMCID: PMC5414512
Scopus ID: 85040642426
  Abstract View: 2370
  PDF Download: 1420

Letter to Editor

Comment on; post-transcatheter aortic valve replacement acute kidney injury; prevention rather than cure

Macaulay Amechi Chukwukadibia Onuigbo 1*, Nneoma Agbasi 2

1 Department of Nephrology, Mayo Clinic Health System, Eau Claire, WI 54702, USA
2 North East London NHS Foundation Trust, UK
*Corresponding Author: *Corresponding author: Macaulay Amechi Chukwukadibia Onuigbo, , Email: onuigbo.macaulay@mayo.edu

Implication for health policy/practice/research/medical education:

There is newly accumulating evidence in the surgery-acute kidney injury (AKI) literature that intraoperative hypotension (IOH) is a formidable and yet preventable causative factor in precipitating postoperative AKI. Postoperative AKI portends both increased short- and long-term morbidity and mortality, increased length of stay and higher healthcare costs. IOH is preventable. We present a case of AKI that led to the syndrome of rapid onset end stage renal disease (SORO-ESRD) in a 73-year-old diabetic hypertensive chronic kidney disease (CKD) male patient in 2012 following minimally invasive surgical aortic valve replacement. We call for more attention to be paid to IOH and to avoid too low blood pressures in the operating room. We posit that systolic blood pressure targets ≥ 90 mm Hg and/or MABP targets ≥ 60 mm Hg potentially would improve postoperative renal outcomes.

Please cite this paper as: Onuigbo MAC, Agbasi N. Comment on; post-transcatheter aortic valve replacement acute kidney injury; prevention rather than cure. J Renal Inj Prev. 2017;6(2):12-15. DOI: 10.15171/jrip.2017.02

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